A postdoctoral or more senior research position is available in the Culotta laboratory at Johns Hopkins University.
The Culotta lab at the JHU school of public health focuses on the role of metal nutrients and reactive oxygen species in fungal biology and fungal pathogenesis. Successful postdoctoral candidates will be trained in bioinorganic chemistry and spectroscopy, and in the molecular and cellular biology of fungal pathogens.
A Ph.D. is required for this position and funding is available for a minimum of two years with possible extensions up to 5 years. The ideal applicant should have a strong academic record, and research experience in molecular biology and/or biochemistry, and fluent English language and writing skills.
To apply, please submit a cover letter, CV, and names of three references that may be contacted. Applications will be reviewed promptly until the position is filled. Johns Hopkins University is an equal opportunity employer.
Culbertson EM, Khan AA, Muchenditsi A, Lutsenko S, Sullivan DJ, Petris MJ, Cormack BP, Culotta VC. Changes in mammalian Cu homeostasis during microbial infection. Metallomics, 2020, Jan 24 DOI: 10.1039/C9MT00294D
Robinett NG, Culbertson EM, Peterson RL, Sanchez H, Andes DR, Nett JE, Culotta VC. Exploiting the vulnerable active site of a Cu-only superoxide dismutase to disrupt fungal pathogenesis. J Biol Chem. Feb 22;294(8):2700-2713. doi: 10.1074/jbc.RA118.007095. 2019
Rossi DCP, Gleason JE, Sanchez H, Schatzman SS, Culbertson EM, Johnson CJ, McNees CA, Coelho C, Nett JE, Andes DR, Cormack BP, Culotta VC. Candida albicans FRE8 encodes a member of the NADPH oxidase family that produces a burst of ROS during fungal morphogenesis PLoS Pathog. Dec 1;13(12):e1006763. doi: 10.1371/journal.ppat.1006763. 2017
Li CX, Gleason JE, Zhang SX, Bruno VM, Cormack BP, Culotta VC. Candida albicans responds to host copper during infection by swapping metal cofactors for superoxide dismutase. Proc Natl Acad Sci U S A. 2015 Sep 22;112(38):E5336-42.
Reddi, A and Culotta, VC. SOD1 integrates signals from glucose and oxygen to repress respiration. Cell. 152(1-2):224-3. 2013.
Tagged as: Life Sciences
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